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The studies here investigate the hypothesis that particulates, including air pollution particles, can induce airway wall fibrosis, a process that can lead to COPD.
The main driving force comes from the reaction products of 10B (n,α) 7Li reaction because both α and 7Li particles can induce soft errors in the memory.
Excessive production of wear particles can induce the osteolysis and then the loosening of the prosthesis components As a tribological design solution to reduce wear, the applications of compliant artificial cartilage materials have been proposed, in which the soft-elastohydrodynamic lubrication is expected to be effective.
Taken together, these results indicate that nanosilica particles can induce allergen-specific Th2-type allergic immune responses in vivo.
Our finding provides a new potential mechanism wherein low toxicity SiO2 particles can induce EPS release from specific phytoplankton, potentially facilitating oil-spill removal by promoting EPS aggregation.
These results suggest that nanosilica particles can induce the production of antigen-specific Ab responses including antigen-specific Th2 allergic immune responses.
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A single alpha particle can induce bystander effects in a population of cells, thus the sensitivity of primary oocytes to low doses of Po noted by Samuels (1966) may be the result of bystander effects.
Alpha emitters can induce DNA lesions in stem cells that result in the transmission of chromosomal instability to their progeny (Kadhim et al. 1992), and even a single alpha particle can induce long-term chromosomal instability in primary human T lymphocytes (Kadhim et al. 2001).
We show that PapMV-CP-M2e virus-like particles (VLPs) can induce production in mice of anti-M2e antibodies that can recognize influenza-infected cells.
However, MOP couplings are associated to the formation of polyethylene wear particles that can induce periprosthetic inflammatory response and osteolysis with subsequent implant failure [ 1– 5].
Those exosomes have an active role in vascular signaling as redox-active particles that can induce endothelial cell caspase-3 activation and apoptosis by generating superoxide, NO and peroxynitrite.
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