Sentence examples for participating brain from inspiring English sources

Recent electrophysiological evidence suggests that the comprehension of emotional prosody and emotional semantics proceeds along two distinct but probably highly interactive and possibly interdependent processing streams which are likely to be subserved by partially overlapping neural networks [9] [11] (and see [12] for review on participating brain structures).

These data show that the interconnectivity between various participating brain areas plays a crucial role in the emergence of pathological oscillations and clinical symptoms.

Evidently, brain contextual processing, from initial perception to subsequent retrieval, is represented not by sequential activation but rather sequential modular communication among participating brain areas.

We hypothesized that the miR-608 multi-target effects would be particularly important for parasympathetic and anxiety-controlling genes participating in brain-to-body communication (4, 14, 16, 17) (Fig.  1A).

All these genes participate in brain development processes such as neuronal migration and axonal guidance, and abnormalities in brain development have been reported in dyslexia (13).

It is also established that most of the resident cells in human brain have the capacity to produce MMP-2 and MMP-9 – enzymes that are known to participate in brain damage (e.g. in case of infectious meningitis) [ 35- 37].

Participants who attended offspring examination 7 (between 1998 and 2001) were invited to participate in brain MRI studies and a neuropsychologic assessment.

Several experimental models of pneumococcal meningitis (PM) have demonstrated that MMPs contribute to brain damage by participating in blood-brain barrier (BBB) breakdown, PMN recruitment, shedding of cytokines as well as their receptors, and cortical damage [ 14- 16].

Richie Sapp knew when he came to Stanford as an undergraduate that he wanted to participate in brain research.

On this basis, the present study was purposely undertaken to (i) define the cellular localization of GPR17 in the intact mouse brain and shed light on its potential roles under physiological conditions; and (ii), to define possible changes of this receptor in the cell types that participate to brain damage and repair in a well-established focal ischemia model such as the MCAo model in the mouse.

These findings suggest that IL-1 α, IL-1 β can be involved in the pathophysiology of atherosclerosis process and also participate in brain injury after the IS.