Sentence examples for ox production from inspiring English sources

The high rate of Ox production with ONOO– is substantiated by the reaction of DNA with SIN-1 that generates NO and O2– that combine to form ONOO–.

The use of a prototypical redox system, i.e. the diffusion controlled reduction of ferricyanide (Ox) and concurrent production of ferrocyanide (Red), allows for a proof of principle evaluation of the viability of SIR for simultaneous mapping (in time and space) of the concentrations of species in the diffusion layer.

To examine whether MED could affect the ox-LDL-induced production of TNF-α, IL-6, and IL-1β in macrophages, the production of these cytokines by ox-LDL-stimulated macrophages was measured in the absence or presence of MED.

We also confirmed that Ox-AT induced production of both IL-8 and MCP-1 in primary NHBE cells.

As shown in Fig.  3a, MED significantly inhibited the ox-LDL-induced production of TNF-α, IL-6, and IL-1β.

Draft power loss due to sickness of oxen, treatment cost, production losses (milk and growth reduction), interference with agricultural activities, induced mortalities and replacement cost were considered as important impact of bovine trypanosomosis for 68.3%, 53.7%, 48.8%, 45.15%, 35.4% and 25.6% of the respondents, respectively.

Interestingly, CAP significantly decreased not only immunoreactivity of OX-42 and OX-6 but also production of microglia-derived reactive oxygen species (ROS) in the SN of MPP+-treated rats.

In this study, we assess the effect of Ox-AT on the production of monocyte chemotactic protein-1 (MCP-1) and IL-8.

There was no significant reduction in Ox-AT-induced IL-8 production in the presence of the MAPK inhibitor (Ox-AT + SB203580, P = 0.379) (Fig. 6 A ).

There was no significant effect on Ox-AT-induced MCP-1 production in the presence of the MAPK inhibitor (Ox-AT + SB-203580) (Fig. 6 B ). A549 cells were treated with either the NF-κB inhibitor (Bay11-7082, 10 μM) or the JNK inhibitor (SP-5600125, 10 μM) dissolved in 10% DMSO followed by 0.03, 0.1, or 0.3 mg/ml Ox-AT (Fig. 7).

Pretreatment with anti-LOX-1 mAb, Vitamin C, apocynin or catalase significantly reduced ROS production and prevented ox-LDL-induced apoptosis, while indomethacin or allopurinol had no effect.