The remaining 12 patients were assigned to the poor outcome group.
Patients were grouped as follows: group A, successful outcome; group B, successful outcome after dynamization; group C, no successful outcome.
After the first VGIR, the clinical outcome was assessed, and 18 patients were assigned to the good outcome group.
Two outcome groups were defined as follows: a "good outcome" group, which contained 6 patients with no serious sequelae at hospital discharge, and a "bad outcome" group, which contained 12 patients who died, had chronic renal failure, or had central nervous system sequelae at hospital discharge.
Good neurological outcome group remained 83% (125/ 150) and poor outcome group 17%(25/150).
Poor outcome group defined, if GCS remained same or even deteriorated than admission.
Good outcome group had fewer episodes of both hyperglycemia and hypoglycemia.
Good outcome group had fewer episodes of brain hypoglycemia during systemic hypoglycemia (p = 0.0026).
In this case, the first stage of the model is a simple binary logit dealing with the selection of individuals into two (latent) groups: a possibly positive outcome group and an always zero outcome group.
The most frequently reported abnormality was a slowed brain electrical activity (22 out of 25 patients in the poor outcome group and 6 out of 9 in the poor outcome group) (P =0 .41).
Glycerol: When compared during the first two days of monitoring, the glycerol levels were higher in the unfavourable outcome group (Mean 220.86; SD 25.85) as compared to the favourable outcome group (Mean 191.76; SD 31.89)[NS].
