Sentence examples for our voltage from inspiring English sources

Choosing our voltage and time units in such a way that C m = g L = 1, we can summarize our approximation to the stochastic differential equation model (1.1) as the evolution given by d V = ( − V + V L + μ c ) d t + σ C d B t (1.3).

Our voltage clamp data show that burst discharges appear to be driven by powerful volleys of glutamate release, whereas tonic spiking is driven by a combination of regular autonomous spiking and irregular glutamate and GABA release, which presumably reduces the regularity of spike discharge.

If we assume release probabilities in the range of ∼0.1B0.3 (consistent with a binomial fit to the distribution of IPSCs observed in our voltage clamp experiments) we would obtain estimates of the expected background rate of uIPSCs in the range of 100B600 Hz, or less if some of the release sites were in severed axons or silent somata.

To inject the patterns of excitatory input we recorded in vivo, we added an AMPA synaptic conductance (Erev = 0 mV) which was varied dynamically to correspond to the conductance underlying our voltage clamp traces, assuming a driving force of 70 mV.

Our voltage clamp trains from −80 to 0 mV vs. −40 to 0 mV, designed to eliminate INa, could also alter NCX function during the contraction train; however, this would promote Ca2+ entry at −40 vs. −80 mV and thus increase Ca2+ waves rather than reduce them.

Our voltage clamp experiments reveal that the locomotion-evoked spiking is driven by a significant increase in excitatory synaptic input to the granule cells, which is paralleled by an increase in spiking in mossy fibers, as shown by direct recordings from mossy fiber boutons.

The membrane potential in the control cells was reduced by ∼5 mV in Gd3+ condition (Figure 5A) but was not affected by Gd3+ in U-type knockdown group, consistent with our voltage-clamp data showing that Gd3+ partially blocked leak current of RPeD1.

Our voltage-clamp experiments allowed us to identify the biophysical properties of a conductance modulated by serotonin in FSIs.

Because of the difficulty in achieving voltage clamp on Purkinje neurons ≥14 days in vitro, our voltage-clamp experiments were performed on the younger cells.

In marked contrast with our results with CA2 PNs, neither our somatic voltage recordings nor phase-plane plots showed evidence of a spikelet.

They ask not about what our electrical voltage is, but whether there is any electricity in our land at all.