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The ELISA analysis used in our study detects C-terminal CCN2 which translates into detection of both C-terminal fragments and whole CCN2.
While our study detects the impact of these events, documented in the observed patterns of admixture, we make no claims as to the timing of population expansion because it is unclear how biases in calling SNPs from GBS data would affect the site frequency spectrum and thus obscure any demographic signal.
MAIR-IR spectrometry used in our study detects such functional groups and most other covalent bonds (Smith 1998) but will not detect inorganic leaching ions from filler particles, which might also affect cells in the proximal vicinity of the resin composite restorations in the mouth.
The 1801 monoclonal antibody employed in our study detects a mammalian-specific epitope of both mutant and wild-type p53 protein.
Further, the accelerometer used in our study detects movement and not posture; the latter function would increase the accuracy to assess sedentary time objectively.
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Our study detected some complex trends in parasite transmission related to lineage sorting within the matrix.
Our study detected a loss of neurons producing gamma-aminobutyric acid (GABA) as their output and glycinergic neurons, along with the pronounced loss of cholinergic neurons.
In addition, our study detected no variation among tissues of an individual (Fig. 1B).
It is important to note that the probe for NTRK2 in our study detected both the full length as well as the truncated forms of NTRK2.
In conclusion, our study detected an association of actinic skin damage with cardiovascular disease, cancer and all-cause mortality in whites.
Our study detected eight SNPs with P < 10−5 (Table III).
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