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The Wnt signaling pathway is activated in CLL, and our data strengthens its role in prognosis.
Our data strengthens the observation that MBP and SUMO partners can be used to promote correct folding of difficult-to-express proteins [ 58, 75].
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Our data strengthen the concept that inflammation arising from cardiac myeloid cells may exert paracrine beneficial impact on cardiomyocytes38,39.
Our data strengthen evidence for a role of this terminal complement component in druse pathogenesis.
Our data strengthen the role of STAT3 as a major player in IL-9 mediated CCL11 expression in ASM cells.
Nevertheless, our data strengthen the hypothesis of alterations in synaptic function underlying the pathophysiology of Rett syndrome.
Using several experimental approaches, our data strengthen the notion that CFTR is involved in the regulation of intestinal FA metabolism through modulation of the lipogenic pathway.
Therefore, our data strengthen the idea of bidirectional inhibitory interactions between B7-1 and PD-L1 in the absence of PD-1 molecules.
Our data strengthen the notion that LRP1 contributes to the pathological nature of this disease, in large part via promotion of EphA2 dependent signaling.
In summary, our data strengthen the link between intergenic transcription and modification of histones over wide chromatin domains, and suggest that developmental regulation of expression the human β-globin genes occurs in part through epigenetic changes to chromatin structural domains.
Taken together, our data strengthen the view that many proteins found in the PV perform an essential function, and highlight the importance of the PV itself for parasite survival.
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CEO of Professional Science Editing for Scientists @ prosciediting.com