Our data describe a proxy for the first of these objectives, but not the second.
Importantly, the list of highly cited researchers was published in 2010, whereas our data describe the current state of Wikipedia, having left sufficient time to Wikipedia editors for a proper inclusion of the listed prominent scientists.
Altogether, our data describe the cellular pathway used by CHIKV to enter mammalian epithelial cells.
Our data describe the intimate interaction between IL-6 and HGF in regulation of inflammation.
Our data describe the efficient production of RSPs containing native prM and E envelope proteins for all dengue serotypes.
Our data describe the relationship between postsynaptic multiple spikes and the naturalistic induction of persistent LTP in the freely behaving rat.
In conclusion, our data describe a differential role for CD80 and CD86 in regulation of inflammation in the innate immune response to sepsis.
Our data described here strongly suggest that in a variety of human carcinomas, over-expression of Ets1 may not just be a prognostic indicator, but play an important causal role that can be exploited for targeted therapy.
Our data describe a novel immunoregulatory property of sHLA-G molecules, based on the inhibition of chemokine-driven migration of different T cell populations together with downregulation of the expression and function of selected chemokine receptors on these cells.
In any case, further study is required to determine the mechanism of coxI initiation, but transcript information provided by Stewart and Beckenback (2009) [55] and our data described below indicate that the Arg (CGR) indeed functions as the initiation codon.
