Exact(7)
Collectively, our data contributes to the rational design of more efficient and less expensive enzyme mixtures, targeting the viable production of bioethanol and other biorefinery products.
Our data contributes to the knowledge that the vast majority of ancient DNA damage is represented by type 2 transitions caused by cytosine deamination and that type 1 transitions could be attributable to PCR artefacts.
Our data contributes to previous evidence suggesting that downregulation or absence of POLQ expression leads to inaccurate DSB repair.
Our data contributes to the global antimicrobial resistance surveillance of N. gonorrhoeae, and emphasizes the importance of further gonococcal antimicrobial resistance testing.
Further, our data contributes to the evidence that a minor allele frequency of ~10% characterizes the SNPs that are associated with FCGR2B gene expression differences.
Therefore, our data contributes to a better understanding of the inflammatory and remodeling processes in both treatments in this experimental model of asthma and may assist researchers to further studies based on their expected outcomes.
Similar(53)
Notwithstanding, our data contribute a preliminary understanding of this population to the research literature.
Our data contribute to dissect the role played by P2 receptor antagonism in sustaining neuroprotection against metabolic stresses.
Despite these limitations, we strongly believe that our data contribute relevant knowledge regarding the roles of SOD as early predictor of AKI in patients with septic shock.
In addition to the doubling of the known mammalian PP1 interactome, our data contribute to the design of PP1 interaction networks.
Our data contribute to the understanding of temporal heterogeneity of HGSOC immune microenvironment and have implications for selection of samples for biomarker testing in the setting of immune-targeting therapeutics.
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