Thus, our data argue against the earlier reported role of the CYP17 in BC predisposition and progression.
Zebrafish Pst is in principle capable of synthesizing PSA, however, our data argue against a fundamental function of the enzyme during development.
Our data argue against genuine between-sex differences in cerebral activation patterns during lexical verbal fluency activities when confounding factors like performance differences are excluded.
Overall, our data argue against the local production of known eicosanoid PPARγ agonists, at least in the prolonged phase of critical illness.
In conclusion, our data argue against an important role of PlGF during primary tumor growth in most models and suggest that clinical evaluation of anti-PlGF antibodies may be challenging.
Our data argue that rAd-infected human DCs genetically engineered to produce IL-10 may be exploited for the modulation of harmful Th1-type responses in transplantation and autoimmune diseases.
Taken together, and similar to the acute treatment of mania with anticonvulsants (Rosa et al. [2011]), our data argue against the idea of a class effect of anticonvulsants in the prevention of depressive and manic episodes in bipolar disorders.
Our data argue that there is direct interaction between Mx2 and HIV-1 CA. Interestingly, several mutations in the HIV-1 viral capsid (CA) region of Gag can overcome MX2-mediated suppression (Fricke et al., 2014; Liu et al., 2013).
In summary, our data argue against the mutually exclusive model proposed for mouse primitive erythrocytes [5] [7].
Hence, our data argue for a critical role of iNKT cells in the immune surveillance of carcinoma that is independent of tumor-specific CTL.
Thus, our data argue for a hierarchical and synergistic relationship between the Hh and Wnt signaling pathways in cigarette smoke-induced lung cancer.
