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Of course, as with all studies, we have to temper our conclusions with limitations.
We demonstrate where affinity and specificity of protein-protein interactions arise across the family, and corroborate our conclusions with extensive experimental evidence.
To be sure, IS-LM is an attempt to squeeze a dynamic economy into a static model, which is why people like me usually cross-check our conclusions with something intertemporal.
As a society, we do a very good job with these kinds of debates: we give everyone a hearing, and pass laws, and make compromises, and square our conclusions with our constitutional heritage — and in the Oregon debate the quality of the theoretical argument was impressively high.
In fairness, the consistent results of three studies should imbue our conclusions with some confidence.
We now report such a case, which was successfully resected, and discuss our conclusions with reference to the current literature.
Similar(33)
Both these approaches lend credence to our conclusion with respect to the paucity of structural disorder in extremophiles.
Given that our conclusion with regards to mitomycin C-mediated inhibition of HIF-1α protein accumulation contradicts a previous study, we wanted to confirm that protein degradation is not involved in the drug effect.
We illustrate our conclusion with the example of coding a patient's expression: can't even go help my sister in the country with potato planting, a bit sad (2).
However, it is acknowledged that our conclusion with induced RR, without further experiments, may not be generalized to naturally observed RR such as those in mouse embryonic stem cells, mouse 3 T3, or rat NRK cells.
As a last study, we studied the robustness of our conclusion with respect to changes in the modeling of input (net uptake) of long-chain fatty acids (LCFA), C16 and C18, in the tricarboxylic acid (TCA) cycle.
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CEO of Professional Science Editing for Scientists @ prosciediting.com