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Moreover, our analysis uncovers the unskilled answerer profile, which has not received much attention in the context of Q&A systems.
Our analysis uncovers the transcriptional circuits that function in establishing distinct mesenchymal compartments downstream of HAND2 and upstream of SHH signaling.
Our analysis uncovers a highly ordered developmental program that correlates with the spore morphological changes and reveals the spatial and temporal molecular events fundamental to reconstruct a cell.
Our analysis uncovers conserved topology-function relationships on a relatively small number of high-level GO terms.
From this, our analysis uncovers further key differences between the population structure of these two species; providing new clues as to the reasons for the persistence, and most recently decline of S. flexneri.
Our analysis uncovers major differences in the regulation of c-myb transcription in the HoxA9/Meis1-transformed cell line compared with the HPC model, which could explain the overexpression of c-myb observed in leukemic cells.
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Our analysis uncovered that the occurrence of these errors is favored by the existence of multiple aromatic or condensed ring systems with a small number of substitutions and the presence of conjugated environments.
But our analysis uncovered something different: The key problem wasn't work-family conflict but long hours across the board.
In our analysis, uncovering correlation is limited to identifying genes that experience similar selective regimes.
Our analysis uncovered a network of transcription factors that potentially dysregulate several genes in MS or one or more of its disease subtypes.
Additionally, our analysis uncovered B3 genes from other families that have underwent recent duplication in different chromosomes, as well as old duplication events (Table S6).
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CEO of Professional Science Editing for Scientists @ prosciediting.com