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Thus in OSA, it refers to a band to probe for instance.
Although sleep architecture is globally preserved in children with obstructive sleep apnoea (OSA), it is considered to be an REM sleep REMS disorder.
Intermittent hypoxia is a major pathophysiologic character of OSA; it can lead to oxidative stress and inflammation, which in their turn cause endothelial dysfunction, a hallmark of atherosclerosis.
Because people who carry extra weight are more prone to OSA, it often helps to drop a few pounds.
In OAm therapy for OSA it has not yet been defined to what extent variations in OAm design may affect clinical efficacy, side effects and patient compliance.
Since, the above study was assessing the PSG of the patients who likely were initially at high risk for OSA, it is impossible to extrapolate this data to general Chinese population.
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It was well established that once UAO in patients with OSA occurs, it requires much higher inspiratory pressure to open the collapsed airway and the end-expiratory pressure requirement to maintain the airway patent is lower after the obstruction is overcome [ 27- 29].
In unselected OSA patients, it is not realistic to expect a clinically relevant decrease in cardiometabolic biomarkers with CPAP therapy.
Treatment with Continuous Positive Airway Pressure (CPAP) offers some protection from the effects of OSA, although it is still unclear which populations should be targeted, for how long, and what the effects of treatment are on different organ systems.
Obesity is a major risk factor for OSA because it directly or indirectly contributes to upper airway narrowing during sleep.
Overnight-attended PSG is an old standard in the diagnosis of OSA, but it is expensive and cumbersome.
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