In many organisms, expression of A-type lamins does not appear until midway through embryogenesis, suggesting a role in differentiation [4], [5].
However, in both organisms, expression of the major nitrite reductases is co-activated by FNR and a two-component regulatory system, NarQ-NarP, that bind to similar target sequences located at almost identical positions relative to the respective transcription start sites (P aniA in N. gonorrhoeae; P nirB in E. coli [ 5, 6, 14- 18]).
While whole cell or whole organism expression of FPs are common, fluorescent reporter genes have also been cloned under the control of tissue-specific promoters for discrete expression in transgenic plants, including in pollen [ 9, 10], endosperm and aleurone cells [ 11- 13], roots [ 14, 15] and vascular tissues [ 16, 17].
Depending on the organism, expression of AOX can be influenced by developmental signals, tissue specificity, and response to stress (Considine et al. 2001; Djajanegara et al. 2002; Finnegan et al. 1997; Karpova et al. 2002; Nargang and Kennell 2010; Van Aken et al. 2009; Vanlerberghe and McIntosh 1997).
Exposure of these cells to cytokines or other factors produced by a variety of cell types infected by HIV (or other pathogenic organisms) activates expression of HIV receptors needed for the infection and replication [29] [32].
This was reminiscent of results in other model organisms where expression of mutant LRRK2 is associated with increased sensitivity to rotenone.
Furthermore, in these organisms, the expression of the U3 snoRNA gene is dependent upon the presence of a closely linked, upstream and divergently oriented tRNA gene [ 44, 45].
As has been suggested across other organisms, temporal expression of validated circRNAs was moderately correlated with that of their linear counterparts [Column Q in Additional file 27] [ 86, 87].
This is also consistent with data from other organisms, where expression of the loading complex must be reduced to near completion to cause cohesion and segregation defects (Heidinger-Pauli et al. 2010).
The organisms alter expression of genes controlled by the farnesoid X receptor (FXR) through bile acids, leading to differential activation by the acids and their metabolites [chenodeoxycholic acid (CDCA) > deoxycholic acid (DCA) > lithocholic acid (LCA) >> cholic acid (CA)].
The expression pattern of CvSEP1, a class E-like MADS-box gene, reflects the expectations resulting from experiments in model organisms; namely, expression of SEP1/2-like genes was expectable for whorls II-IV, although 'expression in sepals is common but not universal' ([ 24], page 431).
