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In many organisms, expression of A-type lamins does not appear until midway through embryogenesis, suggesting a role in differentiation [4], [5].
In diploid organisms, expression from two, potentially different, copies of each gene contribute to transcript level and subsequent protein production.
However, in both organisms, expression of the major nitrite reductases is co-activated by FNR and a two-component regulatory system, NarQ-NarP, that bind to similar target sequences located at almost identical positions relative to the respective transcription start sites (P aniA in N. gonorrhoeae; P nirB in E. coli [ 5, 6, 14- 18]).
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For each organism, expression vectors across tissue types were normalized separately using the R function norm.
For example, Mack et al. (2006) compared their postmating gene expression profiles in the D. melanogaster lower female genital tract to McGraw et al. (2004)'s whole organism expression patterns and found little overlap.
While whole cell or whole organism expression of FPs are common, fluorescent reporter genes have also been cloned under the control of tissue-specific promoters for discrete expression in transgenic plants, including in pollen [ 9, 10], endosperm and aleurone cells [ 11- 13], roots [ 14, 15] and vascular tissues [ 16, 17].
Depending on the organism, expression of AOX can be influenced by developmental signals, tissue specificity, and response to stress (Considine et al. 2001; Djajanegara et al. 2002; Finnegan et al. 1997; Karpova et al. 2002; Nargang and Kennell 2010; Van Aken et al. 2009; Vanlerberghe and McIntosh 1997).
Although Hsp90s are expressed in most organisms, their expression increases in response to stresses.
In all these organisms, boule expression is meiotic.
Exposure of these cells to cytokines or other factors produced by a variety of cell types infected by HIV (or other pathogenic organisms) activates expression of HIV receptors needed for the infection and replication [29] [32].
In Arabidopsis (as in all organisms) HSP expression is strongly induced in response to heat stress.
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