Sentence examples for optimal expression of the from inspiring English sources

Exact(10)

Here health represents a full and optimal expression of the individual's capacities and uniqueness, while illness represents a restricted or limited expression of the self and occurs in response to an interaction between internal and environmental conditions.

The two strains were first grown at 37 °C in stage 1, and then the temperature was transitioned to 20 °C in stage 2 for the optimal expression of the violacein synthesis pathway.

Optimal expression of the CrSPI-1-D1 in bacteria was obtained by induction with 0.5 mM Isopropyl β-D-1-thiogalactopyranoside (IPTG) of 1 liter culture at 25°C.

The conditions like temperature, isopropyl β-D-thiogalactopyranoside (IPTG) concentration, and duration of induction were standardized for optimal expression of the protein in soluble form.

Optimal expression of the CapA-frGFP fusion protein was achieved at 20 °C with 12 15 h duration of the expression induction phase.

Aside from the CUP1 coding sequence of about 200 bp, optimal expression of the gene and copper-induced transcription requires about 300 bp of upstream sequences (Thiele and Hamer 1986).

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It appears then, that while the levels of GnRH-R may be correlated with the optimal expression of each of the subunits [13], simply increasing or decreasing receptor numbers may not actually bring about complete differential gene expression.

These shortcomings notwithstanding, there is a clear pattern that emerges from our experimental data and clinical analysis that strongly suggests that miRNAs regulate the optimal expression of HR proteins in the course of the cell cycle and prevent ectopic activation of the HR pathway.

Taken together, these findings demonstrate that optimal expression of Hes1 counteracts the effects of Aβ on neurons at all the levels examined here.

In cells that were cultured under conditions allowing optimal expression of photosynthetic pigments the development of an intracytoplasmic membrane system was detected by transmission electron microscopy of ultrathin sections (Figure 2D).

Furthermore, because IRF-3/7 play an indispensible role for the induction of the optimal expression of type I IFNs, their delayed activation, relative to NFκB and AP-1, would likely postpone the onset of type I IFN-mediated innate antiviral responses, thereby greatly compromising the ability of the infected host to keep early viral replication in check.

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