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Moreover, ((-1,0)) is the optimal coincidence point of g and T.
Hence (x^{ast}in A_{0}(t)) is the optimal coincidence point of a pair ({g,T}).
However, ((-1,0)) is the optimal coincidence point of g and T, satisfying the conclusion of the theorem.
Moreover, ((-1,0)) is an optimal coincidence point of g and T. Let (T Arightarrow B) be a continuous, proximally monotone, and proximal fuzzy ordered ψ-contraction of type-I.
Moreover, ((0,0)) is optimal coincidence point of g and T. Let (T Arightarrow B) is continuous, proximally monotone, and proximal ordered fuzzy ψ-contraction of type-II, (g Arightarrow A ) surjective, fuzzy isometry and inverse monotone mapping.
A point x in A is said to be an optimal coincidence point of the pair of mappings ((g,T)), where (T Alongrightarrow B) is a nonself mapping and (g Alongrightarrow A) is a self mapping if M gx,Tx,t)=M A,B,t) holds.
Similar(53)
Moreover, our studies provide strong evidence that the NR2B subunit represents a universal rate-limiting molecule for gating NMDA receptor's optimal coincidence-detection property and for enhancing memory function in adulthood across multiple mammalian species.
Moreover, the superior memory function in NR2B transgenic rats, together with the previously reported memory enhancement effects in NR2B transgenic mice [7], [20], [21], provides clear and consistent evidence that the NR2B subunit represents a rate-limiting genetic factor in gating NMDA receptor's optimal coincidence-detection and memory formation in the adult brains of multiple mammalian species.
This approach fails to capture a more encouraging feature of the data: seasonal coincidence of optimal control effectiveness with the peak of actual transmission rather than with mosquito densities.
Thus, NR2 subunits have been regarded as rate-limiting molecules in controlling the optimal NMDA receptor's coincidence-detection property and subsequent learning and memory function across multi-species [6].
One major theory in learning and memory posits that the NR2B gene is a universal genetic factor that acts as rate-limiting molecule in controlling the optimal NMDA receptor's coincidence-detection property and subsequent learning and memory function across multiple animal species.
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