Exact(7)
We were able to make isochrones for the onset of responses based on single sweep optical recordings.
The atypical values we observed at 2 minutes after stimulation were presumably caused by variations in onset of responses in individual cells.
Typically the onset of responses was approx. 2 4 ms following a negative DC stimulation.
As expected, a decrease in the binding rate kon was sufficient to reproduce a slower onset of responses to non-saturating [GABA].
Onset of responses to target/pattern motion was determined as the time at which the mean discharge rate exceeded 2 SD of the baseline value (Fig. 2 C ). Latencies of eye movement responses were measured as the time at which the mean oppositely directed (e.g., upward/downward) eye velocities diverged.
The most apparent difference between α1β2γ2 and α3β2γ2 receptors was a dramatically slower current onset of responses elicited by non-saturating [GABA] in the case of α3β2γ2 receptors (Fig. 1), suggesting a substantially lower value of the association rate constant kon.
Similar(53)
Repeated stimulation of the skin below the level of the lesion, such as rubbing the same area for 20 minutes every day, causes a change in latency (the interval between the stimulus and the onset of response) of certain reflexes, with diminution and finally extinction of the response.
Th1 cells produce IFNγ and facilitate the onset of response against intracellular pathogens, while Th2 cells secrete mainly IL-4 and mediate protection from extracellular microbial agents [5] [7].
The rate of onset of response is slow.
The maximum time for onset of response was 4 weeks.
(E – G ) Differences in the lag times before the onset of response deactivation (Lag Deactivation).
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