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An important aspect of our model is that solicitation has no direct fitness cost during the offspring stage.
Selection does however favor increased solicitation x in the offspring stage, which will increase the amount of obtained PI (fo) if, and only if, parents are sensitive.
Differential selection on these two components of provided PI depends on the level of solicitation expressed in the offspring stage of generation t+1.
In our model, selection on the offspring stage of generation t does not affect parental traits (generation t−1) because these individuals belong to different generations.
Fitness was calculated over both life-stages as the product of survival at the offspring stage and fecundity at the parental stage (i.e., W = S×F).
Analogously, parental provisioning genes, despite only being expressed during the parental stage, must survive the offspring stage before they generate a fecundity pay-off during the stage at which they are expressed (Figure 1).
Similar(42)
We compare variance measurements from human clinical data and from mouse models and show that the mutation level variance is clearly higher in the human data than it is in the mouse models at both the primary oocyte and offspring stages of inheritance.
Our results also showed that the determinants of survival may differ between offspring stages.
For both offspring stages, a social variable best explained variation in survival.
Similarly, in both models, the interaction between an ecological and a social factor exerted a significant effect on offspring survival, but again, our analyses revealed differences in the details of survival determinants between different offspring stages.
Therefore, the transcriptional up-regulation of 'VEGF signaling' reflects the effort on cell maintenance and angiogenic growth in LP offspring at stage 188 dpn.
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