Sentence examples for offspring formation from inspiring English sources
We hypothesize that intracellular offspring formation in Epulopiscium evolved from endospore formation and these two developmental programs share molecular mechanisms that are responsible for the observed morphological similarities.
Clearly further functional studies are required to test the above models and identify additional mechanisms acquired or modified during evolution of offspring formation in Epulopiscium.
Similar results were obtained with or without 5-fluoro-2′-deoxyuridine (FUdR), which inhibits offspring formation in the control (Table 1).
Moreover, the expression pattern of spoIIE in Epulopiscium during offspring formation [ 55] is very similar to that seen in sporulating B. subtilis[ 59].
Given this context, the general picture of the genomic features and DNA organization of genomic regions affected by structural reorganizations is still incomplete, as is that of how genomic changes are transmitted to the offspring during the formation of germ cells and contribute to speciation.
To determine whether these injected cells contributed in spermatogenesis and support the formation of offspring, GFP gene was traced in the genome of rat offspring.
In the case of the pea aphid, Acyrthosiphon pisum Harris (Homoptera: Aphididae), wing formation among offspring is maternally induced when the mother is under adverse biotic conditions, for example, triggered by crowding, low host plant quality, or the presence of natural enemies [19] [28].
This process is indispensable for formation of viable offspring.
The formation of these offspring has been described in stages that parallel stages of endospore formation [ 54- 56].
Although disease recurrence was observed in some families, it was thought that related mtDNA duplications were the actual transmitted molecule, leading to deletion formation in the offspring [29,30].
Notably, gestational stress can dysregulate progesterone formation in juvenile offspring [ 30], a change that may persist into adulthood in female F1 and F2 animals to perturb physiological and behavioural adjustments to pregnancy.
