According to Taatgen and colleagues [6], most individuals tend to protect memory consolidation of a first target by temporarily blocking the detection of subsequent targets, resulting in an AB.
Thus, facial emotional expressions, particularly fear, can be processed in the absence of awareness [5], [6], triggering changes in skin conductance [7], [8] and judgment of subsequent targets [9], [10].
For this reason, Experiment 1 employed briefly presented and masked primes, which the participant was instructed to ignore, and measured the amount of priming (i.e., facilitated naming) of subsequent targets that were either the same object as the prime, or a different object.
It thus seems that acoustic similarity to speech is a prerequisite for the LTAS of the precursors to influence the perception of subsequent targets.
In the absence of Ng, however, the same induction protocols are not able to induce LTP, as there is not enough CaM targeted (spatially regulated) within the spine to allow the proper activation of subsequent targets necessary for LTP induction.
Since Experiment 2 showed that not all precursor signals influence perception of subsequent targets in the same way, an LTAS compensation process that is indifferent to the exact nature of the precursors cannot fully account for whether normalization effects occur.
Experiments 4a and 4b show that reintroducing pitch movement or silent periods in the signal does not change the influence of a precursor sentence on subsequent targets.
This suggested these compounds may act on at least two targets, the first of which caused a growth-slowing or static mode of action, whilst inhibition of the subsequent target(s) had a cytocidal effect.
On the basis of this long-term average, the perceptual impact of acoustic energy in certain frequency regions of a subsequent target sound becomes attenuated.
One possibility is that subliminal repetition suppression reflects short-lived, visual representations of the prime that facilitate perceptual processing of the subsequent target.
