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Mass spectra of TMS derivatives of erythrodiol and uvaol showed a common fragmentation pattern: is spite the molecular ion is minimal or not present, it can be easily deduced by presence of loss of methyl group [M CH3 ] at m/z 571, tipycal of silylated compounds, and a intense neutral loss of the TMSiOH group at m/z 496 that represent the base peak of the spectra of either compounds.
Higher concentrations of either compounds were not well tolerated (Additional file 1: Figure S1).
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A deficiency of either compound leads to disordered production of DNA and, hence, to the impaired production of red blood cells.
No significant cytotoxic effect of either compound was observed.
Significant reductions in overall HIC activity were not observed following administration of either compound.
Acute administration of either compound 12 or 29 resulted in a specific, dose-dependent reduction in food intake in rats.
Then, we consider generically disordered particles made of grains of either compound A, or compound B, with an excess of B grains.
IAPs, the NF-κB downstream genes were down-regulated, and caspase-3 and -9 were activated by the treatment of either compound (Fig. 3).
It has been shown that the formation of either compound was strongly dependent on the concentration, temperature and pH of solutions.
Passive samplers deployed in vials with permeable membrane septa showed no detection of either compound, likely due to lack of water motion in the vials.
These effects were lost if the dose of either compound extended into the range which disrupted performance in the baseline DMTP task.
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