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Current audited circulation is 247,390 and this is expected to fall again in the next round of ABCs due out in February.
The molecular mechanisms that promote the expansion and function of ABCs are largely unknown.
It has been shown that at least a subset of ABCs has a specific cytogenetic aberration, most commonly a translocation [4].
The experimental results on 22 benchmark functions demonstrate that GRO could enhance the exploitation ability of ABCs and accelerate convergence without loss of diversity.
Cummings et al. [9] in a preliminary study found that argon beam coagulation after curettage provided improved local control of ABCs compared with curettage without adjuvant or with phenol and not associated with an increase in the operative complications.
A better understanding of the pathways controlled by the SWEF proteins could help provide new insights into the mechanisms responsible for the expansion of ABCs in autoimmunity and potentially guide the design of novel therapeutic approaches.
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P-gp and BCRP are two members of the superfamily of ABC-transporters.
Inner current loop is feedback control of abc phase current instantaneous values [23].
Lenalidomide treatment of ABC-DLBCL cells resulted in downregulation of SPIB at the level of transcription.
Recent studies revealed that MALT1 is a promising therapeutic target for the treatment of ABC-DLBCL.
The molecule shares the common overall architecture of ABC-transporter NBDs.
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