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In this paper, we present two biomechanic ocular models that are easily integrated into the available musculoskeletal models.
While many simulation models exist for ultrasound training, there are currently no ocular models.
This was demonstrated using the following ocular models of inflammation and angiogenesis: endotoxin-induced uveitis, experimental autoimmune uveoretinitis and laser-induced choroidal neovascularization model.
Angiogenesis has been studied by means of several in vitro and in vivo models, including endothelial cell cultures, chick embryo chorioallantoic membrane (CAM) assay [ 4], and ocular models [ 5].
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Based on the data from a performed in vitro release study, a kinetic ocular model to predict in vivo drug concentrations in a rabbit vitreous was built.
This study utilized the rabbit ocular model of HSV-1 infection and reactivation, induced by the transcorneal iontophoresis of epinephrine (TCIE), to characterize changes to chromatin that occur between 0.5 and 4 h following the application of the reactivation stimulus.
To accomplish these goals, we used the rabbit ocular model of HSV-1 infection and reactivation induced by the transcorneal iontophoresis of epinephrine (TCIE) to identify potential changes in the chromatin profiles of two phenotypically different wild-type HSV-1 strains.
Morrison et al. [ 14] have suggested, however, that pathophysiology in the episcleral vein cautery model differs from the other two ocular hypertension models, and the pattern of RGC death might be different in this model.
This article describes a technique for optical modeling of an ocular prosthesis.
Ocular inflammation models Karsten Gronert; 32.
These Rho-kinase specific inhibitors were applied to rabbit ocular hypertensive models and were shown to reduce intraocular pressure.
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