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As soon as the source observes a delay, it reduces the rate to the initial value.
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We observed a delay in the breakthrough of the early injected water between approximately 0.5 1.0 injected pore volume, at 25 °C only; after that, the oil started to flow.
That's why we observed a delay in osteoclast formation by 5 to 6 days with the bone slices.
We also observed a delay in development in the mtp2 morphants, characterized by a large yolk, decreased embryo size, and decreased melanocyte pigmentation (Figure 6B).
In accordance with the decrease in proliferation and migration in cultured keratinocytes we observed a delay of re-epithelialization in our wound healing model.
In the BcorNeo cells we observed a delay in the downregulation of the ES cell markers Oct3/4 and Nanog, whereas Fgf5 was still up regulated appropriately but its down regulation was delayed by two days.
During the early stages of non-stabilized fracture healing in the absence of MMP13, we observed a delay in initial bone deposition correlated with an increase in cartilage formation.
Because we observed a delay in the growth of U87-EGFRvIII glioma cells co-cultured with hMSC-scFvEGFRvIII, we hypothesized that this effect might be mediated through changes in the activity of downstream signaling molecules, in particular, pAkt.
In summary, this study observed a delay in the onset of T1D as well as physiological and immunological differences in the gut which correlated with the presence of Lactobacillus johnsonii N6.2.
Consistent with this, we observed a delay in T-cell proliferation in response to WNV infection, similar to the observations made in rodents [9] Rather, we believe it likely that the interaction between the specific virus or category of viruses and/or the high viral dose used in all animals may have circumvented any age-related differences and had produced a robust humoral response.
This was further refined by DeWit et al. [32] who observed a delay in progression to harm for the youngest drinkers (ages 10 and under) and identified the age range 11 14 as critical for mediating elevated risk of developing alcohol-related disorders.
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