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One example of response shift can be observed our data on global health status, a scale on general quality of life.
While a non-significant association suggesting RB1 alterations to be linked to therapy resistance was observed, our data do not suggest a major role for RB1 alterations explaining acquired drug resistance.
Once we showed gene expression dynamics of adhesins from A. naeslundii and S. gordonii during oral biofilm formation, we observed our data is in agreement with other in vivo analysis performed by immunodetection of surface molecules by fluorescence labelling and co-localization [ 8].
Since in RA joints a predominance of Th1 and Th17 activity is observed, our data suggest that in joints of RA patients further Th1 and Th17 cell skewing is facilitated by local inflammatory mediators (for example, IL-12) produced, for example, by macrophages.
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In contrast to Ras, activation of the Rac or Rap1 GTPases upon Epo-stimulation was not observed (our unpublished data).
We recorded the number of runs that have as many smaller introns in bats as observed in our data (observation).
We observed that our data fit the range of values observed in literature, with the exception of TAG and SM (Figs. 4 and S5).
In support of this idea, the N1 segmentation index, which was observed in previous segmentation studies of continuous speech [4], was not observed in our data.
In no instance did we observe the trend towards the causal versus the reactive model that was observed in our data.
Although no such transition function is clearly observed in our data, there could be a possibility that future studies find transition functions that exhibit chaotic behaviors.
In our study, three epsin (1/2/3) genes, one AP180 gene, and one adaptin-like gene were observed in our data.
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CEO of Professional Science Editing for Scientists @ prosciediting.com