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Moreover, we also observed inflammation and alpha-synuclein phosphorylation in the ENS and DMV.
Additionally, we observed inflammation and an enhanced cell proliferation.
After 8 weeks of TAA treatment, we observed inflammation and formation of fibrotic septa.
We additionally observed inflammation inferior to the symphysis pubis (see online supplementary figure S1).
The observed inflammation generally improved at later sacrifice points on days 7 21.
Since we observed inflammation in our histologic examination of Kcne2 tm1a/tm1a mutants, we determined the concentration of cytokines in the plasma.
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At the conservative threshold reported here we observed inflammation-dependent activity within the right medial thalamus, encompassing the mediodorsal nucleus.
Thus, failure to observe inflammation in TKO mice cannot be attributed to an intrinsic proliferative defect in the inflammatory cells themselves.
Nevertheless, thermography was used to observe inflammation reaction caused by contaminated growth promotant ear implants in cattle [ 9].
It is not surprising to observe inflammation in the specimens examined, likely due to several factors including the specific anatomic location and an overall inflammatory response of the stroma to the tumor, among others[ 7, 9].
Changes observed included inflammation, cellular inflammation, joint space narrowing, synovial hyperplasia, erosion, and fibrosis.
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