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The extracellular domain of the human prolactin receptor (hPRL-R) was chosen as a target system because of its number of activating hormones [ 5, 6, 8] and the biological implications of being able to selectively tune its activity [ 3, 9, 10].
NK cell activation is controlled by a number of activating and inhibitory receptors [2].
In the present data from BD and TB patients, inhibitory regulation of Vδ2+ T cells was not up-regulated, but the small number of activating receptor NKG2C+ γδ T cells were relatively increased, implicating not a recent activation inducing NKG2A on δ2+ T cells.
However, whereas T cell activation is controlled primarily by a single receptor, i.e. the TCR, NK cells are regulated by the integration of signals from a number of activating and inhibitory receptors [32].
For some components, there may be multiple regulators that are active in one updating event and the combinatory effect is determined by comparing the number of activating to inhibitory factors.
NK cell responses are controlled by a large number of activating and inhibitory cell surface receptors.
Similar(48)
At the high initiator concentration, a large number of activated monomers become available.
Moreover, the gene array results also indicate that a relatively small number of activated genes can be linked to NUP98-Hox induced effects on primitive hematopoietic cell function.
Multiple reports have cautioned against over-interpretation of results of labeled transplanted cells to host cells, though there has been a dearth of reports quantifying the actual number of activated tissue macrophages engulfing the markers or marked cells [24] [29].
The number of activated molecules rapidly decreased nearly to zero after the brief blue light illumination.
In both the SN and HC of PD patients, the number of activated, CD68 positive, amoeboid microglial cells was increased.
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