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In the LP nucleus, source of dense TC projections to PE and PEa, the territories of origin also contain neurons projecting to the premotor cortex, though more to PMd than to PMv.
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The cytoplasm and nucleus sources of these 13 different CMS types are summarized in Table 1, with BT-CMS and Dian1-CMS used in japonica and other systems used in indica hybrid rice production.
Intermixed with them are a lesser number of small cells, which use GABA as a neurotransmitter and project exclusively to the inferior olivary nucleus, the source of climbing fibers.
This integration partly occurs in the vestibular nuclei, the source of secondary neurons destined for the extraocular muscle nuclei of both sides.
There was also tight clustering among the raphe nuclei, the source of serotonin in the CNS [Fig. 5a (red), b (red and green)].
Nucleus sources are either indica (I) or japonica (J).
At the junction of the midbrain and pons, large numbers of AP+ cells populate the dorsal raphe nucleus, a major source of projections to the nucleus accumbens (Figure 5I).
In humans, decreased Dlx1 expression in the mediodorsal thalamic nucleus, the principal source of thalamocortical connections that selectively terminate in layers III and IV of the PFC, is associated with psychosis [30].
Little is known about Dlx expression in adult human brain; however, decreased Dlx1 expression in the thalamic mediodorsal nucleus, the principal source of thalamic afferents to the cortex that synapse on both pyramidal neurons and interneurons [29], has been associated with psychosis [30].
In any signaling systems, it is plausible to imagine that positioning of the nucleus near the source of the signals can facilitate transduction of the signals.
Opiates affect multiple structures that provide major afferent input to the ventral tegmental area (that is, the nucleus accumbens and the ventral pallidum) as primary sources of GABA input; the prefrontal cortex, the amygdala, and the mediodorsal thalamus as primary sources of glutamate input; and the pedunculopontine tegmental nucleus as a source of acetylcholine input.
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