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In conclusion, only negligible changes in the nuclei frequency occur during the three ontogeny stages in the studied organs.
It is interesting to observe that the > 5C nuclei frequency seems to be dose-dependent.
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Furthermore, when the frequency of nuclei aberrantly oriented towards the contralateral side was examined, Wnt5a−/− mice showed a 7-fold increase compared to WT (Figure 7A, C).
Frequency of nuclei with two, one or zero signals for each X-borne locus.
We scored the frequency of nuclei with paired HIM-8 signals in five zones that correspond to the premeiotic stage and four substages of meiotic prophase.
In the premeiotic zone (zone 1), the frequency of nuclei with paired signals is less than 20% in both wild-type and pgl-1 mutant.
Mean copy number of CDKN2A and CEP9 and frequency of nuclei with loss of PIK3CA were significantly different in the CM group compared with normal mucosa and marginal levels of deletions in TP63, FHIT, PIK3CA, EGFR, CDKN2A, YES and gains at PIK3CA, TP63, FGFR1, MYC, CDNK2A and NCOA3 were detected in few CM cases, mainly with dilation grades III and IV.
A fundamental issue in NMR spectroscopy is the estimation of parameters such as the Larmor frequencies of nuclei, J coupling constants, and relaxation rates.
This coil design strategy is advantageous and well suitable for multinuclear MR imaging and spectroscopy studies, particularly in the case where Larmor frequencies of nuclei in question are not separate enough.
Those studies showed that stimulation in various thalamic nuclei evoke short-latency frequency-depend excitatory cortical responses that can augment (spreading significantly when stimulating in VL/VA) or depress (when stimulating in VPM/VPL).
In another investigative approach, frequencies of nuclei with normal (2 copies) or loss (<2 copies) and gain (>2 copies) for each gene were compared (Table 2), and only for PIK3CA there were higher frequencies of cells with copy number loss in the CM group compared to NM (36.4% vs. 28.5%).
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