Sentence examples for nuclear injury from inspiring English sources

Results of the decrease of LDH release and the relief of nuclear injury also indicate the protective effects of pinocembrin on this process.

Loss of HMGB1 in the pancreas increases histone (H3 and H4) release into the circulation after extensive nuclear injury and cell death.

In line with the increased viability, a decrease in LDH release and relief of nuclear injury also indicated the protective effects of quercetin in this process.

Based on the above results, quercetin at 0.3 μmol/L, 3.0 μmol/L and 30.0 μmol/L is found to significantly increase the cell viability, decrease LDH leakage, and ameliorate the nuclear injury induced by fA β1 40.

The toxic effect of fA β1 40 at 20 μmol/L was achieved in hBMECs, involving decreasing cell viability, increasing LDH release, inducing nuclear injury, overproducing intracellular ROS, reducing SOD activity, and breaking the barrier function.

Takashima approached the issue from the standpoint of inferior olivary nuclear injury in his study of 15 premature infants who sustained neuronal loss and gliosis to this structure; he observed "a close relationship between lesions of the inferior olivary nuclei and the presence of cerebellar hemispheric lesions such as cerebellar hemorrhage…" [ 19].

Interestingly, it appeared that electrically-active, conducting cardiomyocytes such as atrioventricular nodal cells exhibited more severe nuclear injuries and developed apoptosis much earlier than the non-conducting counterparts[ 18].

In experiments in the present study, we used LA-QPCR to examine general mitochondrial and nuclear DNA injury, including the formation of bulky adducts and oxidative damage, both of which may be playing a role in BaP-induced carcinogenicity.

In addition, numerous tubular epithelial cells showed membrane injury, nuclear pycnosis and necrosis.

While lutein can alleviate cytotoxic and oxidative injury, nuclear factor erythroid 2-related factor 2 (Nrf2) pathway plays a critical role in defending oxidative species.

These networks, which are multidisciplinary in composition, have a single clinical or disease focus (for example burn injury, nuclear medicine, aged care, stroke) and largely rely on clinicians providing voluntary, often out of hours work.