This functional complexity involves the association with nuclear factors and non-coding RNAs.
EMSA and foot-printing analysis confirmed binding of tobacco nuclear factors with modified TGACG motif/s.
These hepatocyte nuclear factors are transcriptional activators of liver-specific genes, such as albumin and transthyretin, and they also interact with chromatin.
Notably, we also identified many transcription and nuclear factors whose expression profiles were strongly linked to each cell type (Supplementary Data 1).
During periods of starvation, ATXN2 is transcriptionally induced and localized to cytosolic stress granules, where nuclear factors dock to compensate RNA pathology.
This compaction affects DNA structure and coordinates the accessibility to site-specific nuclear factors during DNA processing events.
Stage-specific gene expression is crucial, yet few nuclear factors, including potential epigenetic regulators, have been identified.
Most prominently, Cn targets the nuclear factors of activated T cell (NFATs), transcription factors that activate cytokine genes.
We therefore determined the relationship between the nuclear factors that bind to the TCR delta and Moloney murine leukemia virus core motifs.
SUN1 and SUN2 could affect DDR by localizing certain nuclear factors to the NE or by mediating communication between nuclear and cytoplasmic events.
Immunostimulating cytokines and bacterial pathogens activate iNOS and generate high concentrations of NO through the activation of inducible nuclear factors, including NF-κB [68].
