Sentence examples for nscs it from inspiring English sources

For clinical therapy using NSCs, it is essential to secure a large number of differentiated NSCs.

To establish feasibility of MRI for visualization of FE-Pro labeled NSCs, it was important to determine the in vitro detection level by MRI.

In addition, since two factors are sufficient for human iPS induction from NSCs, it may be possible to substitute one or both of these factors with small molecules that activate OCT3/4 or KLF4 gene expression.

In addition to internalization and retention of the FE-Pro label by NSCs, it is important to establish the sensitivity of MRI FE-Pro-labeled NSCs at a particular resolution with MRI sequence field strength.

While we do not purport to know the molecular mechanisms by which amiodarone HCL leads to the toxicity we observed in NSCs, it is notable that the results of our automated screening, including GSEA and microarray analysis, are all consistent with published literature that implicates the roles of ion channels and TNFα signaling in amiodarone-mediated cytotoxicity.

For striatal NSCs, it means a decline in NSC numbers and neuronal differentiation.

When Ms Rice ran the National Security Council (NSC), it was hopelessly dysfunctional torn asunder by disputes between the hawks (Mr Cheney and Mr Rumsfeld) and the doves (then secretary of state Colin Powell and his deputy, Richard Armitage).Dealing with a feud between such big political beasts was probably too much for anybody, let alone a relatively inexperienced newcomer.

While in the neural stem cell (NSC), it is the stable but not the unstable miRNAs enriched in the function of "Human embryonic stem cell (hESC) regulation" (P = 0.003).

It can induce senescence-like changes in various cell lines but in NSCs its effect remains undetermined.

Our results indicate that Rest ablation did not impair the proliferation of Sox1+/Nestin− NSCs; however it is difficult to assess the proliferation of Sox1+/Nestin+ NSCs, due to the paucity of Nestin+ NSCs derived from REST-null ES cells.

Furthermore, we demonstrated that VEGF-C/VEGFR3 signaling is evolutionarily conserved in human NSCs, where it promotes cell proliferation and activates ERK/AKT pathways.