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Figure 2 Room-temperature PL spectra of NPs in air and dispersed in hexane and squalane.

The cell cultures were exposed to NPs via air phase on the different horizontal distance between them and the cell culture containing the colloidal fluorescent NPs.

Slides were washed in 0.4 × SSC/0.3% NP-40 followed by 2 × SSC/0.1% NP-40, air dried, and counterstained with 125 ng ml−1 DAPI in antifade.

This value is somewhat lower than expected for a spherical Ag NP in air.

Some 3 T3-L1 samples were cultured in DMEM without NPs but, in air contact, were allowed with wells containing NP suspensions.

Exposure modeling strongly suggests that soil could be a major sink of NPs compared to air and water ecosystems [10, 11].

The core shell structured iron NPs show an air stability as long as one month as a result of the protection of oxide shell.

To further verify whether these oxides (NiO and NiOOH) were generated from surface chemical reactions of Ni NPs with the air rather than with the backing Al2O3 film, a 20-nm Ni film was deposited under the same ALD conditions.

HS images were collected for the same NP substrate with air (n = 1.00), water (n = 1.33), or glycerol (n = 1.47) present between the top of the substrate and the cover slip.

Translocation of NPs from the air-blood barrier to the capillary lumen may take place through the degenerated structures with acute inflammatory condition [ 15, 17].

These converging lines of evidence led us to analyze brain levels of Aβ40 and Aβ42 in mice exposed to an inhaled particulate matter (nickel nanoparticle; Ni NP) model of air pollution.