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Samples of different impurity levels or different catalyst loadings (n-nonylphenol (NP), 1 to 10 phr) were studied.
Interactions and signaling through these receptors are also modulated by the co-receptors neuropilin (NP) 1 and 2 [ 6, 7].
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The crossover occurs for NP ~1.
To benchmark our comparisons, we set τ, the effect size of a single risk allele, to be equal to g2 np +1 /g2 np with g2 np +1 and g2 np calculated from the Probit model.
As expected, because HBoV NP-1 is highly conserved, sequence variation among New Zealand isolates and the prototype Stockholm ST-1 and ST-2 (1 ) NP -1 sequences was limited.
Furthermore, COE was neuroprotective against the mitochondrial toxin 3-NP (1 mM) [ 36].
The notochord condenses within the primitive AF to form the NP [ 1, 4].
Simulations suggest that VEGFf activity is controlled by its interactions with NP-1 and VEGFR1 and that release or restriction of NP-1 controls whether enhanced VEGF binding of VEGFR2 is likely to occur.
These results are based on NP-1.
The NP-1 gene expression capability maintained basically the same level.
The fault planes NP-1 and NP-2 included the epicenter.
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