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influenza infection by adoptive transfer of CFSE labeled naïve influenza HA specific TCR-transgenic Clone 4 (CL-4) CD8+ T cells into normal recipients [20], [21].
Our data from embryo transfer also indicated that blastocysts from diabetic mice can implant in the normal recipients, while blastocysts from control mice have a lower implantation rate in diabetic recipients.
The relative risk of VAPP varies by serotype: Immunologically normal recipients are at highest risk from type 3 virus, immunodeficient recipients are at highest risk from type 2, and contacts are at highest risk from type 2. During the period 1997 1999, IPV was given prior to OPV, and in 2000 an exclusive IPV policy was adopted.
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Our data demonstrate that splenocytes from both donor strains cause progressive loss of RGC in normal recipient mice and that T- lymphocytes likely instrumental in this process.
Fetal liver cells (2 × 10 cells) from E14.5 embryos (CD45.2) were injected into lethally irradiated recipient mice (CD45.1) together with bone marrow cells (0.5 × 10 cells) from normal recipient mice.
To test the involvement of cellular immune processes in the pathophysiology of retinal ganglion cell degeneration in vivo, we carried out adoptive transfer experiments from two independent genetic mouse models of glaucoma into normal recipient mice.
To examine the importance of direct effects of leptin on lymphopoiesis and immune response, we generated bone marrow chimeras by transplantation of db/db bone marrow cells into lethally irradiated normal recipient mice, which provide a normal environment for the grafted cells.
One of the earliest findings implicating the putative role of DCs in the breakdown of tolerance was that transferring DCs from normal donor mice to normal recipient mice could cause loss of tolerance in the host, leading to the appearance of anti-DNA and anticardiolipin autoantibodies [ 49].
Therefore, we used national data to compare LOS in select groups of morbidly obese and normal weight recipients after kidney transplant.
After blastocysts from diabetic mice were transferred into normal pseudopregnant recipients, the implantation rate was 89%.
When blastocysts recovered from control mice were transferred into normal pseudopregnant recipients, the implantation rate was 100%.
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