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Histologically, the normal layers of the cerebral cortex were disorganized and, using an immunohistochemical technique against neurofilaments, three layers were identified instead of the six present in normal brains.
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In wildtype mice αSMA staining showed a normal layer of smooth muscle surrounding large airways and blood vessels (Fig. 4G).
Lungs of Creb1−/− mice also showed a normal layer of smooth muscle cells surrounding large airways and blood vessels (Fig. 4H).
Some of the edge-associated vessels retained a normal layer of smooth muscle.
Ruminal contractions were reduced and normal layering of the contents was absent at palpation.
Control slides that were not subjected to any enzymatic treatment revealed PMCs with a normal layer of callose that was thin in the early stages of meiosis and progressively thickened in the final stages (Fig. 1A).
The OCT images showed the loss of tissue texture (absence of normal layers) and presence of pockets of uneven echoic or dark areas, which are caused by high signal absorption in the necrotic tissue or by the presence of the pools of mucus.
The OCT images showed the loss of tissue texture (absence of normal layers).
The epidermis of the SASP−/− mice showed a normal layer organization of keratinocytes, and keratohyalin-granules were observed in both SASP+/+ and SASP−/− mice at the level of hematoxylin eosin (HE -stained paraffin section images (Fig 2A and B).
In Section 2, we describe two-dimensional quasi-variable meshes to deal with parallel and normal layer by means of mesh parameters in the x- and y-directions.
Examination of normal and diabetic hippocampus sections revealed marked effects of diabetes in the form of cell death in several areas, with disruption of normal layer organization.
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