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Dehydration was the cause of one death, hyperthermia another, and two people with structurally normal hearts apparently died of arrhythmias.
This paradoxical motion does not cause any mechanical disadvantage in structurally normal hearts [19].
Surgical patients with limited cardiovascular reserve have much worse prognosis than patients with normal hearts.
Meanwhile, ICA-105574 at higher concentrations showed a potential proarrhythmic risk in normal hearts.
PLSVC occurs in 0.3 % of patients with normal hearts and in 4.5 % of patients with congenital heart disease [1, 2].
In patients with presumably normal hearts, both systolic and diastolic dysfunction with reduced cardiac output were demonstrated by echocardiography during therapeutic hypothermia [10].
In normal hearts, both MPC and IPC significantly reduced infarct size and the rise in lactate dehydrogenase (LDH) level caused by I/R injury.
A structural malformation was present in the majority of cases (54%); however, cardiac pathology in anatomically normal hearts was also common (46%).
Results: Compared with 85 normal fetuses, the a-V and PIV of the fetuses with NT>95th percentile and normal hearts were decreased and increased, respectively.
A high gene expression can be achieved in normal hearts.
Tnnt2+/− mice had normal hearts (histology, mass, left ventricular end diastolic diameter [LVEDD], fractional shortening [FS]).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com