Sentence examples for normal control population from inspiring English sources

Exact(17)

z-scores were derived by comparing the test results to a normal control population.

Wimmer et al. show that malignancy occurs approximately fourfold more frequently in renal-transplant recipients than in a normal control population.

The same quantity discriminated clinically isolated syndrome patients from a normal control population (p < 0.001) and discriminated relapsing remitting from secondary-progressive patients (p < 0.05); hence this new technique may sense early disease-related myelin loss and transitions to progressive disease.

The normal control population consisted in 43 student or staff members.

transplantation recipients showed the prevalence of DRB1*15 (calculated as the proportion of subjects who have either one or two copies of this allele) was greater among the IPF patients than in a normal control population [27] (Table 2).

Thirty healthy donors recruited from laboratory personnel were included as a normal control population.

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Similar(43)

Conversely, the frequencies of DRB1*15 alleles in both the IPF and normal control populations of that previous study [46] were several-fold less than that measured here, and were also much less than frequencies reported within other large Caucasian control populations [22], [24], [29], [30].

The genotype distribution and allele frequencies of the (G-T n microsatellite marker in normal control populations and patient populations are summarised in table 1.

These data suggest future studies should explore the role of gender on WM functioning in both ADHD and normal control populations.

More recently, HD patients were noted to have T cell and NK cell proliferation defects compared to normal control populations; however, potential relationships to IV iron administration were not explored in these studies [ 6, 7].

For example, genetic changes can be detected in CTCs, including abnormal telomerase activity (Soria et al, 1999), allelic loss and/or amplification of multiple oncogenes not seen in normal control populations (Austrup et al, 2000), and aneuploid changes in cellular chromosome content based upon FISH analysis similar to those seen in the primary tumour (Fehm et al, 2002).

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