Sentence examples for non for the development from inspiring English sources

Indeed, loss of contact-dependent inhibition of proliferation is an absolute conditio sine qua non for the development and progression of any solid tumour.

An important feature of atherogenesis is chronic inflammation, a condition thought to be sine qua non for the development of atherosclerotic plaques (Libby, 2006b; Libby et al., 2010).

Because environmental exposure to trauma is the sine qua non for the development of Post Traumatic Stress Disorder (PTSD), the recent focus on genetic studies has been noteworthy.

In any case, these observations suggest that genome duplication is not a sine qua non for the development of evolutionary novelty.

Based on all of these previously published hypotheses, we think that a multifactorial aetiology is the best match with regard to BEN pathophysiology, with a genetic predisposition being the 'condicio sine qua non' for the development of BEN.

The sine qua non condition for the development of innovative companies and the success of the Strategy for Responsible Development are predictability of the legal system and the guarantee of judicial independence.

Numerous studies have provided evidence that, although the transformation of epithelial cells is the sine qua non condition for the development of carcinomas, the nature of the connective/stromal tissue environment is crucial for tumor progression.

Therefore, we believe that the observed changes in fecal microbiota do not represent a sine qua non condition for the development of NEC and are more likely just a symptom of a disturbed homeostasis at the intestinal endothelial barrier.

AR action is a conditio sine qua non for the normal development and function of the entire male genital tract; conversely, varyingly degrees of impaired AR action from mutation is causative in individuals affected by androgen insensitivity syndrome (AIS) [1], [2].

If we regard insulitis as a conditio sine qua non for the later development of type 1 diabetes, given that insulitis is not observed in all cases positive for autoantibodies, we have to consider the possibility that the nature of early disease is remitting relapsing, with phases of intense inflammation alternating with quiescent phases.

In conclusion, the data suggest that both functional LTβR and TNFR2 signaling are required and non-redundant for the development of microvascular pathology resulting in fatal ECM.