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Whole-body NO synthesis was calculated as the product of NO synthesis and body weight.
To clarify whether nitric oxide (NO) synthesis in myomas differs from that in parental human myometrium.
Another potent way regulating NO synthesis is mediated by DDAH.
However, ABA caused closure, dark adaptation induced H2O2 production and H2O2 induced NO synthesis in mutants.
Asymmetric dimethylarginine (ADMA), an endogenous inhibitor of NO synthesis, is extensively metabolised by dimethylarginine dimethylaminohydrolase (DDAH).
Acetylcholine is known to induce NO synthesis via NOS in endothelial cells [ 38].
Furthermore, downregulation of l-arginine transport attenuates insulin-stimulated NO synthesis.
Specifically, PXR activation by xenobiotics appears to up-regulate iNOS and subsequently increases NO synthesis.
Both, basal and cytokine induced NO synthesis influence the effects of β-adrenergic stimulation.
Increased ADMA may limit intracellular L-arginine availability for NO synthesis or inhibit NOS.
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The cause for this is an inhibition of the NO-synthesis by asymmetric dimethylarginine (ADMA).
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