Sentence examples for no stimulate from inspiring English sources

Normal levels of intracellular NO stimulate mitochondrial biogenesis via cGMP and PGC-1 [162] 162].

Normal levels of intracellular NO stimulate mitochondrial biogenesis via cGMP and PGC-1 [ 161, 162].

Both CNP and NO stimulate the synthesis of cGMP and lead to activation of common downstream pathways, involving PKGII.

However, it has also been shown that low levels of NO stimulate mitochondrial proliferation, suggesting that effects of NO on mitochondrial function during sepsis depend on its concentration and the timing of its release [ 35].

NO stimulated measurements were performed in the presence of the NO donor DEA/NO, and NO/YC-1-stimulated enzyme activity measurements were performed in the presence of both DEA/NO and YC-1.

The production of NO stimulated with acetylcholine was greater in 2K than in 2K-1C endothelial cells, and this effect was impaired by methyl-β-ciclodextrin in both 2K and 2K-1C.

These results indicate that after seven days, lead does not seem to modify the release of NO stimulated by acetylcholine or alter NO signaling pathways.

Additionally, NO stimulated the expression of invasion genes, including the transcriptional activator hilA and the invA, invB, invC, sopB, sopE2 and sicA structural and effector components of SPI-1 (table S2).

In P19 cells expressing GFP under a cardiac-specific promoter to monitor their CM differentiation, NO stimulated guanylate cyclase to produce cGMP, an activator of cGMP-dependent protein kinase G [12].

A general increase in antioxidant levels including zeaxanthin, antheraxanthin, lutein and α-tocopherol and a significant rise in NPQ was detected in strain 14H8, where no stimulated O2 production and GPXH expression could be measured any more.

A recent study has shown that production of NO, stimulated by caloric restriction, increases SIRT1 expression; this study suggests that eNOS may be involved in regulation of the expression of SIRT1 in murine white adipocytes [ 17].