Nitric oxide (NO) plays a crucial role in the physiological and pathophysiological regulations of osteoblast functions.
The data indicate that NO plays a key role in insulin-induced hypothermia.
Endogenous nitric oxide (NO) plays important physiological roles in the body.
It has been reported that nitric oxide (NO) plays a role during trigeminovascular inflammation occurring during migraine attacks [2].
Inhibition of terminal respiratory oxidases by nitric oxide (NO) plays important physiological roles in signaling and host defense.
There is considerable evidence that nitric oxide (NO) plays an important role in protecting against atherosclerosis, the process underlying IHD.
Nitric oxide (NO) plays relevant roles in signal transduction in physiopathology and its effects are dependent on several environmental factors.
The present study was designed to test the hypothesis that nitric oxide (NO) plays a role in insulin-induced hypothermia.
The overproduction of nitric oxide (NO) plays an important role in a variety of pathophysiological processes, including inflammation.
The same study proposes that NO plays an obligatory role in eliciting this phenotype.
Nitric oxide (NO) plays an important role in physiological processes.
