Exact(1)
For each simulation, we used MERLIN to generate a new data set with the original phenotypes but with new genotypes simulated under the null hypothesis of no linkage for all autosomal markers, retaining the same allele frequencies, marker spacing, and missing data pattern.
Similar(57)
Here, we have assumed no linkage disequilibrium for simplicity.
We found no linkage disequilibria for any locus combination (Additional file 5).
12 In these studies, no linkage peaks for log SrCr were observed in either of these regions.
We also could not include the Danio KCNA 3-2-10a 3-2-10a 3-2-10ano linkage information for Kclusterasinceilable inothe current assemblinkage
In the present study SCI items could not form a construct together with stroke and TBI since there was no linkage item for a Rasch analysis with items split into diagnosis specific items.
First, there may be no true linkage for these disorders.
* No linkage group obtained for this chromosome.
No linkage disequilibrium occurred for pairs of loci in any population, and Micro-Checker revealed no evidence of stuttering, null alleles, and allele dropout.
No linkage was observed for quinine at the region of chromosome 12 [logarithm of odds, (LOD) = 0.73], but this was to be expected given the lack of statistical power (i.e. with 764 quasi-independent sib pairs and QTL additive variance of 23%, we had 20% power at P = 0.001).
After corrections for multiple comparisons, no linkage disequilibrium was detected for the eight loci employed, but most population samples (23 out of 26) deviated from Hardy-Weinberg equilibrium (HWE) at one to five loci.
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