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Nitric oxide (NO) is involved in the pathogenesis of migraine pain and is formed after cortical spreading depression.
Results support the hypothesis that NO is involved in recruiting to cycle those satellite cells resident on fibers.
These results indicate that NO is involved in spatial recognition memory and that an NO component modulates the effects of the cholinergic system on spatial memory.
The gaseous signalling molecule nitric oxide (NO) is involved in various physiological processes including regulation of blood pressure, immunocytotoxicity and neurotransmission.
Taken together, these results and those of the present study show that a same molecule, NO, is involved in the conduction of excitation along the autonomic nerve fibres and in neuronal communication within the prevertebral ganglia.
So during the functioning of the autonomic networks involved in the organization of the GIR, the same molecule, NO, is involved in both the conduction of excitation without action potentials within the nerve fibres and in the communication between the neurons of the ganglionic networks.
Nitric oxide (NO) is involved in eating behavior and inflammatory response.
This suggests that NO is involved in the development of tolerance to flagelline.
Lastly, nNOS is expressed primarily in the brain where NO is involved in neurotransmission.
Specifically, NO is involved in the dispersal of bacteria from biofilms [ 40].
Previous experimental evidence suggests that NO is involved in the pathogenesis of insulin resistance and diabetes[ 17, 18].
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