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The IgAN patients were next divided into three equal groups according to the tertiles of the uTWEAK distribution: Group 1, uTWEAK levels <72.3 pg/mgCr; Group 2, uTWEAK levels between 72.3 and 123.0 pg/mgCr; and Group 3, uTWEAK levels >123.0 pg/mgCr (Table 2).
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We next divide into four cases to prove.
We next divided patients into two groups: (1) exponential decrease group (group I: 0.9 < R ≤ 1.0), and (2) nonexponential decrease group (group II: 0.9 < R).
We next divided promoters into several non-overlapping regions corresponding to the approximate locations of well-positioned nucleosomes and the nucleosome-depleted region (NDR) (Additional file 1: Figure S2).
We next divided all exons into five bins based on evolutionary conservation levels of the exons among 18 placental mammals (see Materials and Methods).
We next divided the subjects into two groups according to HbA1c (low, <6.5%; high, ≥6.5%) based on the American Diabetes Association criteria [ 19].
Since a substantial fraction of HMR regions found in our methylome maps do not overlap a CGI (72.5% of all fibroblast HMRs and 81.2% of all identified keratinocyte HMRs), we next divided the HMRs into two groups: those that overlap with CGIs and those that do not.
We next divided the losses into three sub-categories: "lineage-restricted losses" (lost in one or two species), "widespread losses" (absent from more than two species, but may have involved more than one loss event), and losses of one paralog of a duplicate gene pair descended from the whole-genome duplication ("duplicate gene losses").
We next divided the data set into subsets, considering the fact that the large number of aftershocks that do not meet the criterion to be used in stress tensor inversion is due to spatial or temporal variations in the stress field.
We next divided 46 single experiments into 3 groups depending on the dynamics observed in each experiment.
We next divided CXCR4-positive CRCs into nuclear and cytomembrane types and further investigated the importance of CXCR4 immunostaining patterns (Table 4).
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