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We here investigated the behavioural and neuronal relationship between the attribution of personal relevance, reward and personality.
The general aim of our study was to investigate the behavioural and neuronal relationship between the attribution of personal relevance and reward.
Although previous research demonstrated a relationship between reward and personal relevance, their exact neuronal relationship including the impact of personality traits remains unclear.
Concurrent measurement of the somatosensory and prefrontal cortices would further reveal the neuronal relationship between primary and higher centers of sensory perception in the pathophysiology of unexplained occlusal discomfort.
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Our data demonstrate that sister neurons within the optic tectum have significantly more similar RF centers than nonsisters, indicating that neuronal lineage relationships influence the fine-scale topography of the retinotectal map.
Indeed, even if the nonlinear neuronal generator relationships (as indicated by Naka and Colleagues [ 61– 65] for bipolar to ganglion cells) manifest in ERG gains, the linear approach provides a close approximation over the short ranges used in our model (~±50%).
Analyzing how sensory signals affect the spiking activity of a neuron requires a good description of the neuronal stimulus-response relationship.
Furthermore, within the same time frame, it reveals the existence of some neuronal classes whose relationship to motoneurons and possible role in locomotor network remains to be defined.
Previously, we identified serotonin (5-HT) as an extracellular signal that can promote axonal mitochondrial movement in cultured hippocampal neurons, suggesting a possible relationship between neuronal activity and mitochondrial movement [9].
The vast number of dendritic geometries, combined with heterogeneous properties of the cell membrane, continue to challenge scientists in predicting neuronal input-output relationships, even in the case of sub-threshold dendritic currents.
In particular, we aimed at assessing the relationship between neuronal injury and inter-neuronal α-syn propagation.
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