Sentence examples for neuronal class from inspiring English sources

Simultaneous recordings between synaptically connected L1 neurons and L2/3 pyramidal neurons (n = 384) demonstrated neuronal class specificity in both excitatory and inhibitory connectivity and the properties of synaptic potentials.

CMI: Cell Migration Index; NSE: Neuron Specific Enolase; TuJ1: Neuronal class III β-tubulin; NF68: Neurofilaments 68; Microtubule-Associatedciated Protein 2; GFAP: Glial Fibrillary Acidic Protein; GAPDH: Glyceraldehyde-3-Phosphate Dehydrogenase; DAPI: 4′,6-Diamidino-2-Phenylindole.

Nearly all c-MYC and MYCN over-expressing cells, as revealed by GFP, translocated into the mantle zone of the neural tube and differentiated into neurons, as revealed by staining for neuronal class III β3-tubulin (Tuj1) and NeuN (Fig 1J M, P-Q, T; Supplementary Fig S3D M).

Neuronal class III beta-tubulin.

Neuronal class III β-tubulin/synapsin I (a and a′), class III β-tubulin/neural cell adhesion molecule (b and b′), and class III β-tubulin/phosphorylated neurofilament proteins (c and c′). Figure adapted from [16].

MiR-130a, microRNA-130a; miR-130a-inh, microRNA-130a-inhibitor; MECP2, methyl-CpG-binding protein 2; TUNEL, TdT-mediated dUTP nick end labeling; Tuj1, neuronal class III β-tubulin.

In addition to the above mentioned neuronal classes, neurons expressing the virus at 40 h postinjection were found in more dorsal laminae (Fig. 4, Fig. 5).

We found that kal-1 mutant animals do not display obvious defects in the overall organization of the nervous systems, nor in a diverse panel of individual neurons and neuronal classes (Table 2).

Since worms possess different numbers of neurons in the various neuronal classes examined, comparative loss across different subtypes was represented in Fig. 2D by the percentage of neurons scored within a given subclass that were degenerating (instead of percentage of worms displaying at least one degenerating neuron, as reported in Fig. 2C).

We demonstrate that these methods are amenable to analysis in both individual neuronal classes and individual neurons.

This metabolite, which disrupts UPS function, caused gradual degeneration of all neuronal classes examined, however DA neurons were particularly vulnerable to exposure.