Sentence examples for neuronal breast from inspiring English sources

Since PTBP1 has a major role in alternative splicing mechanism, its depletion in all three type of cells (Neuronal, breast and ovarian) provides an advantage to determine the accuracy of PTBP1 common and tissue-specific deregulated mechanisms.

This is carried out both at molecular level through structural and kinetics assays applied to the purified enzyme, and at cellular level using neuronal and breast cancer cell lines.

However, NO has been positively correlated with tumour grade in human gynaecological, breast, neuronal, and head and neck tumours (Thomsen et al, 1994; Cobbs et al, 1995; Gallo et al, 1998; Reveneau et al, 1999).

We suggest that metabolic adaptation and increase in the mitochondrial number in prostate cells may result in resistance to the oxidative stress and pro-apoptotic signals initiated by hormone therapy, as seen in neuronal cells and breast cancer xenografts (Besada et al, 2006; Busija et al, 2008; De Simoni et al, 2008).

It is noteworthy that under the influence of a static 0.2 T field, the cell viabilities of different line cells (such as human neuronal FNC-B4 and breast carcinoma cells) are unaffected, even at more intense fields (1.5 T) the growth of HeLa cells is unaltered [59].

Nuclear-to-cytoplasmic relocalisation of BAG-1M has also been observed during epidermal and neuronal differentiation and during breast epithelial involution, both in vitro and in vivo (Takayama et al, 1998; Schorr et al, 1999; Kermer et al, 2002).

These two lists of mechanisms serve as derived Gold Standards to compare their robustness across studies, methods, and underpinning biology (PTBP1 depleted cells; mouse versus human, neuronal versus cancer cell lines; breast versus ovarian cancer cell lines).

Recently, we and others have described a role for HOXC11 in the transcriptional regulation of S100beta in breast cancer and neuronal tissue (Zhang et al, 2007; McIlroy et al, 2010).

The role of the homeobox genes in the transcriptional regulation of S100beta in breast tumour and neuronal tissue (Zhang et al, 2007; McIlroy et al, 2010) suggests their potential for regulating this calcium-binding protein in melanoma.

48 49 The mechanisms underlying this association are unclear; the relationship may reflect socio-economic disadvantage, parental aspirations, early attachment, neurological difficulties or a direct role of breast milk in neuronal development.

Although the less studied physiologic estrogens E1 and E3 have weak effects on transcription (Kuiper et al. 1997; Lippman et al. 1977), they are as effective as E2 in causing ERK phosphorylation and other nongenomic responses in pituitary, breast, uterine, and neuronal cells (Alyea and Watson 2009b; Kabil et al. 2008; Mermelstein et al. 1996; Raz et al. 2008; Watson et al. 2008; Zhang et al. 2009).