There are many situations where unexpected compartmentalization negatively impacts reservoir development.
It is also of special interest what extra effect – be it positive or negative – can be brought about by the compartmentalization of replicators by membranes [ 7, 22, 29, 30] or by inorganic materials [ 31].
One property that Gerhart and Kirschner identified is the compartmentalization of phenotypic effects to avoid negative side effects of genes.
While it is customary to adjust p-values for multiple comparisons when making positive claims, our use of uncorrected p-values is conservative with respect to the negative conclusions in this study (i.e., that compartmentalization was minimal).
The latter refers to a tendency in the narratives to keep positive aspects of the relationships separate from the negative aspects, which we analysed as a form of compartmentalization. Accordingly, the following discussion is divided into the following four sections: (1) Power strategies, (2) Coping strategies, (3) Turning points, and (4) Compartmentalization.
Animal findings and results of human sepsis are complementary, and they suggest a compartmentalization of lipid peroxidation in systemic infections by MDR gram-negative bacteria.
The complementary findings between the described animal model and human sepsis are highly indicative of an existing compartmentalization of lipid peroxidation in systemic infections with MDR gram-negative bacteria.
In these forests, the main preventive action consists of forest compartmentalization by fuel break networks, which entail high costs and sometimes significant negative impacts.
The lack of severe cardiovascular dysfunction and tissue hypoperfusion found in our model, likely due to a compartmentalization of the infection, and swift hemodynamic support, could partially explain our negative findings.
In line with these pieces of evidence, compartmentalization of mTORC1 at the level of the lysosomes could provide some explanations regarding mTOR negative regulation on autophagy.
